Stable Angina

Stable angina, also known as exertional angina, is the most common form. In people with this condition, the coronary arteries are narrowed due to a buildup of plaque. During physical activity, the narrowed arteries have a difficult time getting enough oxygen, which, in turn, causes symptoms such as chest pain. However, with rest and/or medication, the condition generally improves.

What Is Stable Angina?

More than 6 million Americans live with angina pectoris, or angina for short. Angina is chest pain or discomfort that occurs when the heart muscle is temporarily not getting enough oxygen-rich blood. A bout of angina is not a heart attack, but it means that you're more likely to have a heart attack than someone who doesn't have angina.
 
There are three types:
 
 
Stable angina is the most common type; it is also referred to as exertional angina.
 

Understanding the Heart and Coronary Arteries

In order to understand the cause of stable angina, it is often helpful to understand the heart and the coronary arteries. Like any muscle, the heart needs a constant supply of oxygen and nutrients, which are carried to it by the blood in the coronary arteries. Similar to other muscles, the harder the heart is working, the more oxygen and nutrients it needs. However, the coronary arteries can become narrowed or clogged, which can decrease the amount of blood that goes to the heart muscle. When the coronary arteries cannot supply enough oxygen-rich blood to the heart, angina symptoms can occur.
 

Causes of Stable Angina

In stable angina, a coronary artery has been severely narrowed due to the buildup of plaque. Severely narrowed arteries may allow enough blood to reach the heart when the demand for oxygen is low (such as when you are sitting). With exertion, like walking uphill or climbing stairs, however, the heart works harder and needs more oxygen, which it cannot get. Therefore, a person develops symptoms of angina. With rest, the symptoms of an angina attack generally improve (see Causes of Angina for more information).
 
Written by/reviewed by: Arthur Schoenstadt, MD
Last reviewed by: Arthur Schoenstadt, MD
Last updated/reviewed:
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